K009372P

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  • 英文名称
    Anti-SARS spike glycoprotein S1 Polyclonal Antibody
  • 宿主
    Rabbit
  • 别名
    E2;E2 glycoprotein;Human coronavirus spike glycoprotein;Peplomer protein;S;S glycoprotein;SPIKE_CVHSA;Severe acute respiratory syndrome spike glycoprotein;Severe acute respiratory syndrome virus spike glycoprotein;Spike glycoprotein;VGL2. SARS-CoV
  • 应用
    ELISA IHC-P IHC-F ICC IF
  • 稀释比例
    ELISA 1:500-1000. IHC-P 1:100-500. IHC-F 1:100-500. ICC 1:100-500. IF 1:100-500
  • 蛋白分子量
    138kDa
  • 保存
    Store at -20°C. Avoid freeze / thaw cycles.
  • 储存液
    Buffer: PBS with 0.03% sodium azide, 50% glycerol, pH7.4.
  • 纯化方法
    Affinity purification
  • 亚型
    IgG
  • 免疫原
    KLH conjugated synthetic peptide derived from SARS spike glycoprotein:101-200/1255
  • Swiss Prot
    P59594
  • 背景资料
    S1 attaches the virion to the cell membrane by interacting with human ACE2 and CLEC4M/DC-SIGNR, initiating the infection. Binding to the receptor and internalization of the virus into the endosomes of the host cell probably induces conformational changes in the S glycoprotein. Proteolysis by cathepsin CTSL may unmask the fusion peptide of S2 and activate membranes fusion within endosomes.
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  • 英文名称
    Anti-SARS spike glycoprotein S1 Polyclonal Antibody
  • 宿主
    Rabbit
  • 别名
    E2;E2 glycoprotein;Human coronavirus spike glycoprotein;Peplomer protein;S;S glycoprotein;SPIKE_CVHSA;Severe acute respiratory syndrome spike glycoprotein;Severe acute respiratory syndrome virus spike glycoprotein;Spike glycoprotein;VGL2. SARS-CoV
  • 应用
    ELISA IHC-P IHC-F ICC IF
  • 稀释比例
    ELISA 1:500-1000. IHC-P 1:100-500. IHC-F 1:100-500. ICC 1:100-500. IF 1:100-500
  • 蛋白分子量
    138kDa
  • 保存
    Store at -20°C. Avoid freeze / thaw cycles.
  • 储存液
    Buffer: PBS with 0.03% sodium azide, 50% glycerol, pH7.4.
  • 纯化方法
    Affinity purification
  • 亚型
    IgG
  • 免疫原
    KLH conjugated synthetic peptide derived from SARS spike glycoprotein:101-200/1255
  • Swiss Prot
    P59594
  • 背景资料
    S1 attaches the virion to the cell membrane by interacting with human ACE2 and CLEC4M/DC-SIGNR, initiating the infection. Binding to the receptor and internalization of the virus into the endosomes of the host cell probably induces conformational changes in the S glycoprotein. Proteolysis by cathepsin CTSL may unmask the fusion peptide of S2 and activate membranes fusion within endosomes.